iosr-JDMS   Volume-13 ~ Issue-2 ~ February-2014

Abstract:We report here a case of 38 years old healthy male, who presented in the eye OPD with blackish mass over conjunctiva which was increasing in size since 1year. On local examination the mass was on the bulbar conjunctiva near the limbus, darkly pigmented, 0.3 x 0.3 cm size, irregular margins with uneven surface and mobile over the sclera. Other ocular examinations were normal. General investigations were done. The mass was excised under peribulbar local anaesthesia and sent for histpathological examination. The report showed moderate to severe nuclear atypia with evidence of pategoid spread. The subepithelial fibro collaginous tissue shows focal areas with moderate lymphocytic infilteration.

[1.] Seregard S. Conjunctival melanoma. Surv Ophthalmol. 1998; Jan-Feb;42(4):321-50.
[2] Folberg, R, Mclean IW, Zimmerman LE. Malignant melanoma of the conjunctiva. Hum Pathol 1985;16(2): 136–143.
[3]. De Woof- Rouendaal D. Conjunctival melanoma in the Netherlands:a clinico-pathological and follow –up study. [Thesis ] Katwijk, All in BV,1990.
[4] Seregard S, Kock E. Conjunctival melanoma in Sweden 1961-91. Acta Ophthalmol 1992;70:289-296.
[5] Tuomaala S, Kivelä T. Metastatic pattern and survival in disseminated conjunctival melanoma: implications for sentinel lymph node biopsy. Ophthalmology. 2004 Apr; 111 (4):816-21.
[6] Jakobiec FA, Folberg, R, Iwamoto T. Clinicopathologic characteristics of premalignant and malignant melanocytic lesions of the conjunctiva. Ophthalmology 1989; 96: 147-166.
[7] Shields JA, Shields CL, dePotter P. Surgical management of conjunctival tumours. The 1994 Lynn B, McMahan Lecture. Arch Ophthalmol 1997; 115: 808-815.
[8] Tatla T, Hungerford J, Plowman N et al. Conjunctival melanoma: The role of conservative surgery and radiotherapy in regional metastatic disease. Laryngoscope 2005; 115: 817-822.

Paper Type	:	Research Paper
Title	:	The changing trends in antibiotic resistance pattern in septicemia.
Country	:	India
Authors	:	Pavani Nimmala, Parveen Anjum, V.Aruna Rajkumari
	:	10.9790/0853-13230508

Abstract:Septicemia is one of the important causes of morbidity and mortality. Emergence of multidrug resistant bacteria is a major problem in the management of septicemia. Present study was undertaken to isolate the common bacterial pathogens associated with septicemia and determine their antibiotic resistance. Blood cultures from 100 suspected cases of septicemia were examined. The growths from the subcultures were identified by conventional biochemical tests. Antibiotic susceptibility testing done by Kirby-Bauer disk diffusion method and antibiotic resistant strains in the primary screening were further processed for MRSA, ESBL and MBL production.

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Paper Type	:	Research Paper
Title	:	Hyperbaric 2% Lignocaine In Spinal Anaesthesia – An Excellent Option For Day Care Surgeries
Country	:	India
Authors	:	Dr. Sweety Purushotham N, Dr. Abhin Devdas, Dr. Pradeep Javedar, Dr. A.V.Mallikarjuna
	:	10.9790/0853-13230913

Abstract: Hyperbaric Lignocaine is a popular spinal anaesthetic agent especially in short duration surgeries because of its faster onset, intense sensory and motor block of moderate duration and recovery profile. Lower concentrations of hyperbaric lignocaine are used for this purpose. Sixty ASA physical status I & II patients, both male and female posted for various elective surgeries under spinal anaesthesia admitted in our hospital were studied. The patients were given 2% hyperbaric lignocaine 2ml for spinal anaesthesia using 25 G Quincke's spinal needle after uniform pre-medication and pre-loading with 500ml lactated Ringer's solution iv fluid

[1] T.N Calvely & N.E William, Principles & Practice of pharmacology for anaesthetists-5th Ed (Blackwell Publishing, 2009)
[2] Dahl V, Gierloff C, Omland E, Raeder JC. Spinal, Epidural or propofol anaesthesia for out-patient knee arthroscopy? Acta Anaesthesiol Scand, 41(10), 1997 Nov, 1341-5.
[3] Punj J, Khan RM. Spinal anaesthesia for pelvic surgery: low concentrations of lignocaine and bupivacaine are effective with less adverse events. Middle East J Anesthesiol, 22(1), 2013 Feb, 71-7.
[4] Williams N, Doyle A, et al. Spinal anaesthesia for transurethral surgery: comparison of 2% lignocaine with hyperbaric 0.5% bupivacaine. Br J Anaesth. 75, 1995, 9-11.
[5] Harbers JB, Stienstra R, et al. A double blind comparison of lidocaine 2% with or without glucose for spinal anesthesia. Acta Anaesthesiol Scand. 39(7), 1995 Oct, 881-4.
[6] Kristensen J, Helbo- Hansen HS, Toft P, Hole P. Spinal anaesthesia with glucose-free 2% lignocaine. Effect of different volumes. Acta Anaesthesiol Scand. 33(1), 1989 Jan, 53-7.

Paper Type	:	Research Paper
Title	:	Cervical spine Injury clearance protocol in Polytrauma patients
Country	:	India
Authors	:	Dr. Rajesh Ambulgekar, Dr. Pritesh Kothari, Dr Prajyot Kandolkar, Dr. Rohit Takkar
	:	10.9790/0853-13231416

Abstract: It is very common in General Hospital that cervical spine injury is missed in Polytrauma patients at the time of admission. In case where there is no clear indication of cervical spine injury, however, patients still need to be evaluated for cervical spine injuries because an unstable cervical spine injury could to delayed & result in devastation neurological deterioration. So our purpose of study is to reduce the rate of missed cervical spine injury by proposing the clinical guideline for clearance of cervical spine injury.

Keywords: cervical spine injury, neurodeficit, M.R.I., Polytrauma, protocol.ur

[1]. Dunham CM, Brocker BP, Collierr BD, et al. Risks associated with magnetic resonance imaging and cervical collar in comatose,
blunt trauma patients with negative comprehensive cervical spine computed tomography and no apparent spinal deficit. Critical
Care. 12:R89. Epub, 2008.
[2]. Ajani A., cooper D., Optimal Assessment of Cervical Spine Trauma in Critically ill patients : A prospective Evaluation. J Trauma
1998; 26:487-491
[3]. Stiel IG, Wells GA, Vandemheem KL et al. The Canadian C-Spine rule for radiography in alert and stable trauma patients. JAMA
2001; 286(15):1841-1848.
[4]. MacDonald RL, Schwartz ML, Mirich D, et al. Diagnosis of Cervical Soine injuries in motor vehicle crash victims: how many x-rays
are enough? J Trauma 1990; 30(4): 392-397.
[5]. Buckholtz RW et al. Rockwood and Greens Fractures in Adults. Seventh Edition. 2010. Chapter 41- Principles of Spine Trauma
Care.

Paper Type	:	Research Paper
Title	:	EnterococcalInfections And Its Antimicrobial Resistance With Special Reference To VRE And HLAR In A Tertiary Care Hospital In Eastern India
Country	:	India
Authors	:	Ghosh(Ray)Reena, Chatterjee Sumanta, Bhattacharya Kumkum, Bhattacharya Sujata
	:	10.9790/0853-13231722

Abstract: Enterococci have traditionally been regarded as low grade pathogen , have emerged as an increasingly important cause of nosocomial infections in the last decade.Despite increasing reports of VRE in different countries ,there is scanty data on this issue from india especially in the Eastern zone. A total of 157 enterococci were isolated from various clinical specimens (urine,pus& wound swabs,blood and throat swabs body fluids), received in the Microbiology laboratory of a tertiary care hospital in eastern India during the period between July2010 to June2011.

[1]. Desai PJ, Pandit D, Mathur M, Gogate A. Prevalence, identification and distribution of variousspecies of enterococci isolated from clinical specimens with special reference tourinary tract infection in catheterized patients. Indian J Med Microbiol 2001; 19: 132-7.
[2]. Murray BE. The life and times of enterococci. ClinMicrobiol Rev 1990;3:46-65.
[3]. SoodSeema, MalhotraMeenakshi, Das BK, KapilArti.Enterococcal infections & antimicrobial resistance. Indian J Med Res 2008; 128: 111-121.
[4]. Edwards DD, Enterococci attracts attention of concerned microbiologists.ASM News 2000;66: 540-5.
[5]. Cetinkaya Y, Falk P, Mayhall G, Vancomycin resistant enterococci. Clin Microbial Rev 2000;13: 686-707.
[6]. Jones RN, Sader HS, Erwin ME, Anderson SC and Enterococcus Study group. Emerging multiple resistant enterococci. DiagnMicrobiol Infect Dis 1995; 21: 85-9

Paper Type	:	Research Paper
Title	:	Hereditary Hypophosphatemic rickets with Hypercalciuria: A case report and review of literature
Country	:	India
Authors	:	Subal Ku.Pradhan, Pawan Mutalik, Sandeep Ku.Tripathy, Arakhita Swain
	:	10.9790/0853-13232327

Abstract: Hereditary hypophosphatemic rickets with Hypercalciuria (HHRH) is one of the four entities of hereditary hypophosphatemic rickets, characterized by vitamin D refractory rickets, Hyperphosphaturia, Hypercalciuria, hypophosphatemia, elevated serum 1, 25-dihydroxyvitamin D levels, normal serum Calcium, 25-hydroxyvitamin D and PTH levels. Clinically, it is difficult to differentiate the four entities without a genetic test. However, presence of Hypercalciuria by a simple bedside urine examination of spot calcium-creatinine ratio differentiates HHRH from other three entities of hereditary hypophosphatemic rickets. Correction of all the abnormalities,

[1]. Imel EA, Carpenter TO. Rickets: The skeletal disorders of impaired calcium or phosphate availability. Pediatr Endocrinol. 2013; 2: 357-378.
[2]. Jagtap VS, Sarathi V, Lila AR, Bandgar T, Menon P, Shah NS. Hypophosphatemic rickets. Indian J Endocrinol Metab. 2012 Mar; 16(2):177-82. Doi: 10.4103/2230-8210.93733.
[3]. Alizadeh Naderi AS, Reilly RF. Hereditary disorders of renal phosphate wasting. Nat Rev Nephrol2010;6:657–65.10.1038/nrneph.2010.121.
[4]. Carpenter TO. The expanding family of hypophosphatemic syndromes. J Bone Miner Metab2012;30:1–9.10.1007/s00774-011-0340-2.
[5]. The ADHR Consortium Autosomal dominant Hypophosphataemic rickets is associated with mutations in FGF23. Nat Genet 2000;26:345–8.10.1038/81664.
[6]. Feng JQ., Ward LM., Liu S., Lu Y., Xie Y., Yuan B., et al. Loss of DMP1 causes rickets and osteomalacia and identifies a role for osteocytes in mineral metabolism. Nat Genet 2006;38:1310–5.10.1038/ng1905.

Paper Type	:	Research Paper
Title	:	Plasma Cell Mastitis mimicking as carcinoma of the breast: A case report and review of the literature
Country	:	India
Authors	:	Amarendra Prasad Naragam, Shekhar Y Tati, Harish Dara, Ashok Kumar V, Mabu Jada, Prema Kawath
	:	10.9790/0853-13232831

Abstract: Plasma Cell Mastitis (PCM) is an uncommon chronic inflammatory disease of the breast in middle aged women. Patient presents with symptoms of acute mastitis with an abscess or abscesses. Later persists as diffuse lump with nipple discharge. Discharge can be milky, purulent or bloody with axillary lymphadenopathy. Clinically it is very difficult to differentiate from carcinoma (Ca) breast. Microcalcification on mammography makes further confusion in differentiating from carcinoma

[1]. Cutler, M. "Plasma-cell mastitis. Report of a case with bilateral involvement". BMJ, 1949;14:94-96.
[2]. Rodman, J. S., and Ingleby, H. "Plasma-cell mastitis". Ann Surg, 1939;109:921-930.
[3]. Brown KL, Tang PH: Postlactational tumoral granulomatous mastitis: a localized immune phenomenon. Am J Surg 1979,138:326-329.
[4]. Adair, F. E. "Plasma-cell mastitis" A lesion simulating mammary carcinoma: A clinical and pathologic study with a report of ten cases". Arch Surg, 1933;26(5):735-749
[5]. Gaston, E. A. "Plasma cell mastitis" Surgery, 1947; 21, 208.

Paper Type	:	Research Paper
Title	:	Anaesthesia for a Child with Down syndrome Undergoing Cleft Palate Repair and Review of Anaesthesia Management of Other Associated Congenital Syndromes
Country	:	India
Authors	:	Amarjeet D.Patil, Amrita Nandi, Vishwas Sathe, Rakesh Singh, Kanha Agrawal, R.L.Gogna
	:	10.9790/0853-13233239

Abstract: Cleft lip/palate are the most common craniofacial anomalies in children, with an incidence of 1:800 live births. Cleft palate alone occurs in 1:2000 live births1.It occurs due to the failure of fusion or break in fusion of nasal and maxillary processes with the palatine shelves, which form during 8th week of the embryonic period. About 150 syndromes may be associated with cleft deformities. The most well-known are the Pierre Robin's, Treacher Collins and Goldenhaar syndrome. Congenital heart disease (CHD) occurs in 5-10% of these patients1.Surgical repair of cleft lip is usually done at 1-3 months of age for cosmetic purpose and cleft palate at 6 months to 1 year of age to promote facial growth and the speech. The successful outcome following cleft repair depends on the age of the patient, associated morbidities, anaesthetic expertise and post-operative care1.

[2]. Sandberg DJ, Magee WP, Jr, Denk MJ. Neonatal cleft lip and cleft palate repair. AORN J.2002;75:490–8.
[3]. Desalu I, Adeyemo W, Akintimoye M, Adepoju A. Airway and respiratory complications in children undergoing cleft lip and palate repair. Ghana Med J. 2010;44:16–20.
[4]. Takemura H, Yasumoto K, Toi T, Hosoyamada A. Correlation of cleft type with incidence of perioperative respiratory complications in infants with cleft lip and palate. Paediatr Anaesth.2002;12:585–8.
[5]. Nargozian C - The airway in patients with craniofacial abnormalities. PaediatrAnaesth, 2004; 14:53-59.
[6]. Carvalho B. Down's syndrome p212-214. In: Oxford Handbook of Anaesthesia. Ed: Allman KG, Wilson IHOxford University Press 2003
[7]. Roizen MF. Anesthetic Implications of Concurrent Diseases.p974 In: Anesthesia Ed: Miller RD. ChurchillLivingstone 2000. 5th edition;.

Paper Type	:	Research Paper
Title	:	Histopathologic Evaluation of the Subcutaneous Tissue Response toThree Endodontic Sealers
Country	:	India
Authors	:	Dr. Mini.K.John
	:	10.9790/0853-13234049

Abstract: The materials used for obturation of root canal system may get extruded through apical foramen into the periapical tissue. Therefore, biocompatibility of these materials is very important. The purpose of this study was to evaluate the in vivo biocompatibility of three endodontic sealers: Seal apex,Diaket and Tubli seal after their subcutaneous implantationin rats. Each of the materials was injected subcutaneously in the dorsal connective tissue of 20 Wistar albino rats. Tissue biopsies were collected at first day, fifth day, tenth day and thirtieth day after the procedure.

[1]. Serge Bouillaguet,John.C.Watahaetal,Cytotoxicity and sealing properties of four classes of endodontic sealers evaluated by succinic acid dehydrogenase activity and confocal laser scanning microscopy,European Journal of Oral Sciences,112(2),2004,182-87.
[2]. M Aminozarbian M D,E Balimani M D etal,Histopathological evaluation of subcutaneous tissue response to endodontic sealers,Dental Research journal , 2(2), 2006,64-7.
[3]. Huang T H ,Ding S H,Cytotoxicity of resin-zinc oxide eugenol and calcium hydroxide on pdl cells and permanent V79 cells,Int Endo J.35(2),2002 ,153-8.
[4]. Ingle J I.Ingles Endodontics.6thed.Connecticut.Pmph USA ltd :2008
[5]. TorabenejiadM,Walton R E,Endodontics.Prnciples and Practice-Philadelphia,W B Saunders Co, 2009.
[6]. Sanjeev Tyagi ,PriyeshMishra,Evolution of root canal sealers:An insight story,European Journal of General Dentistry ,2(3),2013,199-218.

Paper Type	:	Research Paper
Title	:	Non Surgical Management of Large Periapical Lesions-A Report of Two Cases
Country	:	India
Authors	:	Dr. Bhosale Swati, Dr.Priya Philip , Dr.M.Ramesh Kumar, Dr.Jayasree.S
	:	10.9790/0853-13235054

Abstract: Diseases of dental pulp, if not managed within appropriate time, cause periapical infection which may result in the formation of periapical lesions. It is a general belief that large periapical lesions will not heal by nonsurgical endodontic treatment and needs surgical intervention. But there is no direct evidence that all large periapical lesions need surgical intervention. When endodontic treatment is performed to accepted clinical standards, a success rate of around 90% can be expected. This article highlights the healing potential of non surgical endodontic treatment in the management of large periapical lesions.

Keywords: Calcium hydroxide, Non surgical management, Periapical lesion, Triple antibiotic paste.

[1] Ørstavik D. Time course and risk analysis of the development and healing of chronic apical periodontitis in man. Int Endod J 1996;29:150 –5.
[2] Friedman S. Prognosis of initial endodontic treatment. Endod Topics 2002;2:59–88.
[3] Barbakow FH,Cleaton-Jones PE,Friedman D.Endodontic Treatment of teeth with periapical radiolucent areas in a general dental practice Oral Surg 1981;51:552-9.
[4] Sjogren U, Hagglund B, Sundqvist G, Wing K. Factors affecting the long-term results of endodontic treatment. J Endod 1990; 16: 498–504.
[5] Lin LM,Huang GT,Rosenberg PA Proliferation of epithelial cell rests,formation of apical cysts and regression of apical cysts after periapical wound healing.J Endod 2007;33:908-16.
[6] Nicholls E. endodontics 3rd ed.Bristol :John Wright Sons Ltd 1984 p-206.
[7] Kvist T, Reit C. Postoperative discomfort associated with surgical and nonsurgical endodontic retreatment. Dental Traumatol 2000;16:71– 4.

Paper Type	:	Research Paper
Title	:	Study on Variation in the Origin of Renal Artery.
Country	:	India
Authors	:	Mr.Kishwor Bhandari, Dr. Aruna Mukherjee, Mr. Prakash Mane, Mrs. Sanju Acharya
	:	10.9790/0853-13235557

Abstract: With the increasing numbers of vascular reconstructions and various surgical and radiologic techniques the knowledge of the variations of renal artery has grown its importance. The present study was carried out on 129 renal arteries. The origin of renal artery with reference to lumbar vertebrae was studied. The most frequent position of origin of renal artery was found at the level of L1 which was 55.81%, L1&L2= 32.56%, L2=11.63%.

Key words: Renal artery, abdominal aorta, vertebral level L1..

[1]. Snell R.S. Clinical anatomy 7th ed. Philadelphia; USA: Lippincott Williams & Wilkins.2004.p. 283
[2]. Sing Indirbir. Text book of anatomy vol 2, 5th ed. New Delhi; India: Jaypee Brothers; 2011.
[3]. Gary AT, Kevin TP. Anatomy & physiology 2nd ed. USA:Mosby;1993.
[4]. Tortora GJ, Anagnostakos NP. Principles of anatomy and physiology 6th ed.
[5]. NewYork:Harper Collins Publishers.p. 832.
[6]. Sahana SN. Human anatomy Vol II, 3rd ed.Calcutta:K.K.Publishers;1980. P.

Paper Type	:	Research Paper
Title	:	Primary Non-Hodgkin Lymphoma of the Breast- A Rare Case Report
Country	:	India
Authors	:	Pradeep kumar L, Deepak M B
	:	10.9790/0853-13235860

Abstract: Primary breast lymphoma is a rare tumor that presents commonly as a large mass with an incidence of only 0.5%. It has no specific mammographic appearance and can be confused with poorly differentiated carcinoma on routine FNAC or frozen section. With appropriate treatment, this breast malignancy has a relatively good prognosis. We report a case of primary mammary non-Hodgkin lymphoma in a post-menopausal woman, initially misdiagnosed as a case of ductal carcinoma on FNAC. Subsequently mastectomy specimen revealed non-Hodgkin lymphoma, which was confirmed by Immunohistochemistry.

Key words: Primary breast lymphoma, Immunohistochemistry

[1]. Uesato M, Miyazawa Y, Gunji Y, Ochiai T. Primary non-Hodgkin lymphoma of the breast: report of a case with special reference
to 380 cases in the japanese literature. Breast Cancer, 12, 2005, 154-8.
[2]. Nigam A, Singh AK, Singh SK, Singh N. Primary Mammary(Non-Hodgkin) Lymphoma of Breast: A Case Report. J Cancer Sci
Ther, 3, 2011, 173-5.
[3]. Pullen CM, Cass AJ. Bilateral primary lymphoma of the breast . Aust N Z J Surg, 66, 2000, 845-7.
[4]. Jeon HJ, Akagi T, Hoshida Y, Hayashi K, Yoshino T. Primary non-Hodgkin maligant lymphoma of the breast. An
immunohistochemical study of seven patients with breast lymphoma in Japan. Cancer, 70, 1992, 2451-59.
[5]. Fischer MG, Chideckel NJ. Primary lymphoma of the breast. Breast, 10, 1984, 7-12.
[6]. Mattia AR, Ferry JA, Harris NL. Breast lymphoma(a B-cell lymphoma) of mucosa associated lymphoid tissue. Am J Surg Pathol
17, 1993, 574-87.

Paper Type	:	Research Paper
Title	:	A Study of the Clinical Profile of Cerebral Venous Sinus Thrombosis
Country	:	India
Authors	:	Dr Vishwanath K, Dr K P Balaraj, Dr. B D Chavan
	:	10.9790/0853-13236166

Abstract: Background and objectives: CVT is an important cause of a stroke. Its varied presentations make the clinician to have strong degree of suspicion and investigate accordingly thus instituting early treatment. Objectives: To study clinical profile of cortical venous sinus thrombosis and to assess if possible any prognostic factors.

[1]. Mehta et al.editors. abstracts of 59th annual conference of API:2004Jan18-21:Hyderabad:JAPI{51}2003.P1196
[2]. Agarwal et al. editors. Abstracts of 59th annual conference of API:2004Jan18- 21:Hyderabad:JAPI{51}2003.P12-79 `
[3]. Ameri CA,BousseMG.cerebral venous thrombosis.Neuralclinic1992;10:87-111
[4]. KalbaghRM,Woolf AL,editors.cerebral venous thrombosis.London: university press;1967
[5]. Nagaraja D,Taly AB,puerperal cerebralsino venous thrombosis.progress in clinical neuro science1986;165-177

Paper Type	:	Research Paper
Title	:	Periodontitis And Cerebrovascular Disease
Country	:	India
Authors	:	Prejna Nair, Dr.Radhika Arjunkumar
	:	10.9790/0853-13236769

Abstract: Systemic health is often associated with the condition of the oral cavity because many systemic diseases manifest in the mouth. Effects of systemic disorders on periodontal diseases are well established. Periodontal disease affects many systemic diseases like cardiovascular diseases, diabetes, pre-term low birth weight babies, preclampsia, respiratory infections, cancer, rheumatoid artritis, gastrointestinal disease , osteoporosis, renal disease, prostatitis, alzheimer's disease and cerebrovascular disease. With an increasing body of epidemiological and ecperimental work, specific risk factors and risk indicators for periodontis such as tobacco smoking,demographic factors,and socio-econonomic status have been identified and acknowledged.Any changes to the systemic health because of any of these factors can be seen as a change in periodontal health.

[1] P. Lopez- Jornet,J.D Berna-Mestre,J.D Berna-Serna,F.Camacho-Alonso,S.Fernandez-Millan and M. Reus-Pintado Measurement of Artherosclerosis Markers in Patients with Periodontitis:a case control study: J Periodontal 2012;83:690-698
[2] Snophia Suresh,Satya Narayana,P.Jayakumar,Uma Sudhakar and V.Pramod Evaluation of anti-inflammatory effect of statins in chronic periodontitis : Indian journal of pharmocology.2013jul-aug;45(4):391-394.
[3] Minkle Gulati,Vishal Anand,Nikil Jain, Bhargavi Anand, Rohit Bahuguna, Vivek Govila and Pavitra Rastogi Essentials of Periodontal Medicine in Preventive Medicine: Int J Prev Med.2013 September;4(9):988-994
[4] Ghizoni J.S, Luis antonio de Assis Taveira,Gustavo Pompermaier Garlet,Marcos Flavio Ghizoni,Jefferson Ricardo Pereira,Thiago Jose Dionisio,Daniel Thomas Brozoski,Carlos Ferreira Santos, Adriana Campos Passanezi Sant'Ana Increased Levels of Porphyromonas Gingivalis are Associated With Ischemic and Hemorrhagic Cerebrovascular Disease in Humans:an in vivo study : J.Appl.Oral Sci. Vol.20 no.1 Bauru Jan./Feb.2012

Paper Type : Research Paper Title : Abfraction Myth or Reality Country : India Authors : Dr Pragya Tripathi, Dr Deepak Chopra, Dr Sukhchain Bagga : 10.9790/0853-13237073

Abstract: Abfraction is the loss of tooth structure at the cervical region from heavy occlusal forces. It is described as one of the causes of lesions found along the cervical margins of teeth. This article critically reviews the literature in favour and against the theory of abfraction. From the literature there is little direct evidence supporting the theory of abfraction, apart from laboratory studies, to indicate that abfraction exists other than as a hypothetical component of cervical wear.

[1]. Lee WC, Eakle WS. Possible role of tensile stress in the etiology of cervical erosive lesions of teeth. J Prosthetic Dent 1984;52(3):374-80.
[2]. Grippo JO. Abfractions: a new classification of hard tissue lesions of teeth. J Esthet Dent 1991; 3(1):14-9.
[3]. Grippo JO, Simring M, SchreinerS. Attrition, abrasion, corrosion and abfraction revisited. JADA 2004; 135(8):1109-18.
[4]. Bartlett D.W, Shah P. A critical review of non carious cervical (wear) lesions and the role of abfraction, erosion and abrasion. J Dent Res 2006; 85(4):306-12.

Paper Type	:	Research Paper
Title	:	Ovarian tumours- Incidence and distribution in a tertiary referral center in south India
Country	:	India
Authors	:	Dr.M.Yogambal, Dr. P. Arunalatha, Dr.K.Chandramouleeswari, Dr.V.Palaniappan
	:	10.9790/0853-13237480

Abstract: Ovarian neoplasms have become increasingly important not only because of the large variety of neoplastic entities but more so because they have gradually increased the mortality rate due to female genital cancers. Diverse histopathologies are common in ovarian tumours which reflect its different cell of origin. A total of 7492 gynaecology cases were received at the Department of Pathology, Stanley Medical College, Chennai, India, during the period of January 2009 to December 2013.

[1] Parveen S, Ilyas N, Asghar S. Patterns of care for ovarian cancer: Patients at Institute of Nuclear Medicine and Oncology (INMOL) Lahore. Specialist J Pak Med. Sci 1999;15:209– 15
[2] Murad A. Ovulation induction and ovarian tumours: the debate continues. J Pak Med Assoc 1998;48:353-6.
[3] TortoleroL, Mitchell FM, Rhodes HE. Epidemiology and screening of ovarian cancer. Obstet Gynaecol Clin North Am 1994;21:63–75.
[4] Day N.E, Krishnan E. Epidemiology of gynaecological cancers. Gynaecology by Shaw R W. 2nd ed. Edinburgh: Churchill Living Stone, 1997; p. 477–87.