iosr-JDMS   Volume-17 ~ Issue-3 ~ March-2018

Abstract: Background: Postoperative nausea and vomiting (PONV) are commonly observed undesirable and potential complications in patients after laparoscopic cholecystectomy (LC) and chemotherapy. PONV are commonly observed adverse effects of general anaesthesia (GA) and its incidence ranges between 60% to 72 % following laparoscopic cholecystectomy. Methods: In a prospective, double blinded, randomized controlled study, and after Ethical Committee years who underwent elective LC under GA were studied during a two year period in a tertiary care teaching hospital, in Imphal were assigned in one of two groups viz: Group PD (n=50) received 0.075 mg palonosetron with 8 mg dexamethasone and group GD (n=50) received granisetron 40 μg/kg body weight plus 8 mg dexamethasone. Nausea, retching.......

[1]. Naguib M, Bakry AK, Khoshim MHB, Channa AB, Gammal M, Gammal KE et al. Prophylactic antiemetic therapy with ondansetron, tropisetron, granisetron and metoclopramide in patients undergoing laparoscopic cholecystectomy: a randomized, double-blind comparison with placebo. Can J Anaesth 1996;43(3):226-31.
[2]. Wang JJ, Ho ST, Liu YH, Lee SC, Liu YC, Liao YC et al. Dexamethasone reduces nausea and vomiting after laparoscopic cholecystectomy. Br J Anaesth 1999;83(5):772-5.
[3]. Nesek-Adam V, Grizelj-Stojic E, Rasic Z, Cala Z, Mrsic Z, Smiljanic A. Comparison of dexamethasone, metoclopramide and their combination in the prevention of postoperative nausea and vomiting after laparoscopic cholecystectomy. Surg Endosc 2007;21(4):607-12.
[4]. Rahman MH and Beattie J. A study of post operative nausea and vomiting. Pharmaceutical Journal [serial online] 2008;273:786. Available at URL:http:// www.pharmaceutical-journal.com/learning/learning-article/post-operative-nausea-and-vomiting/10030469.article. Accessed August 10,2015.
[5]. Tramer MR. Treatment of postoperative nausea and vomiting. BMJ 2003;327(7418):762-3...

Abstract: Background: Pediatric ophthalmic disorders are seen to occur because of events that occur during prenatal or postnatal period or childhood. Knowledge about causes and prevalence of ocular morbidity in children is vital for planning and evaluation of preventive and curative services for children. . This study was undertaken to study the pediatric ocular morbidity profile in the inpatient department of a tertiary health care hospital in Goa. Methods: A 6 months retrospective study was conducted from January 2017 to June 2017 on all inpatient pediatric population of ophthalmology department in a tertiary health care hospital in Goa. Data was collected from the clinical records which included detailed history and Comprehensive ophthalmic examination findings. Results: 41 children were admitted......

Keywords: Ocular injuries, Childhood blindness

[1]. Zelalem Addisu Mehari, Pattern of childhood ocular morbidity in rural eye hospital, Central Ethiopia. BMC Ophthalmology201414:50.
[2]. Desai S, Desai R, Desai NC, et al. School eye health appraisal. Indian J Opthalmol 1989; 37: 73-75.
[3]. WHO. Factsheet. Visual impairment and blindness, August 2014.
[4]. Biswas J, Saha I, Das D, Bandyopadhyay S, Ray B, Biswas G.Ocular morbidity in children at a tertiary hospital in Kolkata, West Bengal. Indian J of public health,2012;56:293-6.
[5]. Zahra KB, Shakeel A, Asad AK. Spectrum of ophthalmic diseases in children of a referral hospital. Pak J Ophthalmol. 2007; 23....

Abstract: Introduction- Dyslipidemia, which is associated with hypertension, has been recognized as independent risk factor for cardiovascular disease, a leading diagnosis for visits to physicians and cause of death. Age and gender differences also affect serum lipids considerably.Hypertensive subjects frequently have higher cholesterol levels than normotensive subjects.Though no specific pattern of dyslipidemia has been consistently reported among hypertensive individuals, many studies have shown that total cholesterol (TC), triglycerides (TG), and virtually all fractions of lipoproteins tend to be more frequently abnormal among hypertensive patients than in the general population. Aims and Objectives-To assess the correlation between hypertension and various parameters of Dyslipidemia..............

Keywords: Dyslipidemia, TC, LDL, TG, HDL,Hypertension

[1]. W. B. Kannel, W. P. Castelli, T. Gordon, and P. M. McNamara, "Serum cholesterol, lipoproteins, and the risk of coronary heart disease. The Framingham study," Annals of Internal Medicine, vol. 74, no. 1, pp. 1–12, 1971.
[2]. R. R. Williams, S. C. Hunt, P. N. Hopkins et al., "Familial dyslipidemic hypertension. Evidence from 58 Utah families for a syndrome present in approximately 12% of patients with essential hypertension," Journal of the American Medical Association, vol. 259, no. 24, pp. 3579–3586, 1988.
[3]. R. O. Halperin, H. D. Sesso, J. Ma, J. E. Buring, M. J. Stampfer, and J. M. Gaziano, "Dyslipidemia and the risk of incident hypertension in men," Hypertension, vol. 47, no. 1, pp. 45–50, 2006.
[4]. C. Borghi, "Interactions between hypercholesterolemia and hypertension: implications for therapy," Current Opinion in Nephrology and Hypertension, vol. 11, no. 5, pp. 489–496, 2002.
[5]. J. D. Neaton and D. Wentworth, "Serum cholesterol, blood pressure, cigarette smoking, and death from coronary heart disease: overall findings and differences by age for 316 099 white men," Archives of Internal Medicine, vol. 152, no. 1, pp. 56–64, 1992.

Abstract: In a developing country like India, which is endemic for tuberculosis, patients presenting with intestinal obstruction are often misdiagnosed as abdominal tuberculosis. Similarly, Crohn's disease with increasing incidence is being treated with anti tubercular therapy leading to late diagnosis with increased morbidity and mortality. I am hereby presenting a case series of 3 cases who were being mistreated as intestinal tuberculosis and were later diagnosed as Crohn's disease after surgical intervention. Such cases require a deep study, so that undue intervention and anti tubercular therapy can be avoided

[1]. Fabrizio Michelassi & Roger D. Hurst. Alessandro Fichera. (2012). Crohn's Disease. Zinner J. Michael & Ashley W. Stanley (Ed.), Maingot's Abdominal Operations(665-691). Mc Graw Hill.
[2]. Dalziel TK. Chronic intestinal enteritis. BMJ 1913;2:1068â€―1070.
[3]. Marshall JB . Tuberculosis of the gastrointestinal tract and peritoneum. A m J Gastroenterol 1 993; 8 8: 9 89 – 99 .
[4]. Kirsch R , Pentecost M , H all PM et al. Role of colonoscopic biopsy in distinguishing between Crohn's disease and intestinal tuberculosis. J Clin Pathol 2006 ; 59 : 840 – 4.
[5]. Riedel L , Segal I , Mohamed AE et al. Th e prolonged course of gastrointestinal tuberculosis. J Clin Gastroenterol 1 989; 1 1: 6 71 – 4 .

Abstract: Among HIV-HCV co-infected patients all patients cannot afford the treatment of HCV. The aim is to study the natural history of disease in HIV-HCV co-infected patients on Anti Retroviral Therapy( ART) who could not take treatment for HCV. We hypothesized that Anti Retroviral Therapy will help in decreasing the disease progression of HCV and there will not be much difference in prognosis who are additionally treated with anti HCV drugs. Methods: 145 patients are included in this study who are HIV HCV co-infected who are on ART. Only 40 patients has been treated for HCV in some point of their life. We compared the immunological profile of two groups and paired t test was used to compare it. CD4 count was calculated by Fluorescence Activated Cell sorter.......

Keywords: HIV-HCV Co-infection,treatment,CD4 count,HCV RNA.

[1]. Alexander J. AIDS. In: Kasper DL, Fauci AS, Longo DL, Jameson JL, Loscalzo J, editors. Harrison principle of internal medicine. 18th ed. New York: MC Graw Hill; 2012.p.1215-18.
[2]. Franciscus A. Guide to HIV-HCV Co-infection. Available on www.hcvadvocate.org/hepatitis/factsheets-pdf/Coinfection/20Guide.pdf. Accessed Oct 15, 2015. [3]. Ghany M, Strader D, Thomas D, Seeff L. Diagnosis, management, and treatment of hepatitis C: an update. Hepatology. 2009;49(11):1335–74.
[4]. Resino S, Bellon J, Asensio C, Micheloud D, Miralles P, Vargas A, et al. Can serum hyaluronic acid replace simple non-invasive indexes to predict liver fibrosis in HIV/Hepatitis C coinfected patients. BMC infect dis. 2010;10(1);864-8.
[5]. Eyster ME, Diamondstone LS, Lien JM, EhmannWC, QuanS, Goedert JJ. Natural history of hepatitis C virus infection in multitransfused hemophiliacs: Effect of co-infection with human immunodeficiency virus. The multicenter hemophilia cohort study. Journal AIDS 1993;6(1):602–10.

Abstract: Background: Carpal Tunnel Syndrome (CTS) can be associated with any condition that causes pressure on the median nerve at the wrist. Some speculate that carpal tunnel syndrome is provoked by repetitive movement and manipulating activities and that the exposure can be cumulative. The relationship between BMI, body fat and CTS is due to increased fat deposition in the carpal canal or higher hydrostatic pressure in the carpal tunnel in obese subjects compared to normal or slender individuals. CTS will be common in those who are frequent users of the keyboard, but in societies where livelihood is dependent on agricultural activities, it will be common in those who have to perform manual labour...............

Keywords: Carpal tunnel syndrome, BMI, Hydrostatic pressure, Cross sectional type

[1]. McBride, David. "Carpal Tunnel Syndrome (CTS)." Australasian Musculoskeletal Medicine 19, no. 2 (2015): 19.
[2]. Silverstein B, Fine L, Armstrong T 1987. "Occupational factors and carpal tunnel syndrome‟. American Journal of Industrial Medicine; 11 (3): 343-358.

[3]. Walker JA 2010. " Management of patient with carpal tunnel syndrome‟. Nursing Standard; 24 (19): 44–48.
[4]. Uemura T, Hidaka N Nakamura H 2008. "Clinical outcome of carpal tunnel release with and without opposition transfer‟. J Hand Surg Eur ; 35 (8): 632–6.
[5]. Atroshi I, Gummensson C, Johnsson R et al 1999. "Prevalence of carpal tunnel syndrome in a general population‟. JAMA; 282: 153-8.

Abstract: Sudden onset sensorineural hearing loss is a medical emergency and exact cause is not known in 90 % of cases. Systemic corticosteroids is used in clinical practice as a primary treatment, although there is doubt regarding its efficacy. Several other forms of treatment have been studied, such as plasmaexpanders, carbogen, antiviral therapy, hyperbaric oxygen, antioxidants. The objective of studywas to evaluate clinical features of idiopathic sudden onset sensorineural hearing loss and its outcome on patients after treatment with oral steroids, antiviral and methylcobalamine injection . A non randomized control trial consisting of 70 cases over a period of 2 years were taken. Efficacy of treatment was based on relief of symptoms, pure tone average pre and post treatment. Hence study proved to be effective treatment option for idiopathic sudden onset sensorineural hearing loss

Keywords: Sudden onset sensorineural hearing loss, corticosteroids, pure tone average

[1]. Vijayendra H, Buggaveeti G, Parikh B, Sangitha R. Sudden Sensorineural HearingLoss: An Otologic Emergency. Indian J Otolaryngology Head Neck Surg 2011;64(1):1-4.
[2]. Wilson WR, Byl FM, Laird N. The efficacy of steroids in the treatment of idiopathicsudden hearing loss: a double-blind clinical study. Archives of Otolaryngology1980;106(12):772-6.
[3]. Penido ND, Cruz OL, Zanoni A, Inoue DP. Classification and hearing evolution ofpatients with sudden sensorineural hearing loss. Brazilian Journal of Medical andBiological Research 2009;42(8):712-6.
[4]. Oh JH, Park K, Lee SJ, Shin YR, Choung YH. Bilateral versus unilateral sudden sensorineural hearing loss. Otolaryngology-Head and Neck Surgery 2007;136(1):87- 91
[5]. Bogaz EA, Suzuki FAB, Rossini BAA, Inoue DP, Penido NO. Glucocorticoid influence on prognosis of idiopathicsudden sensorineural hearing loss. Braz J Otorhinolaryngol. 2014;80:213-9.

Abstract: The syndrome of painful ophthalmoplegia consists of periorbital or hemicranial paincombined with ipsilateral ocular motor nerve palsies, oculosympathetic paralysis, and sensory loss in the distribution of the ophthalmic and occasionally the maxillary division of the trigeminal nerve. Various combinations of these cranial nerve palsies may occur, localizing the pathological process to the region of the cavernous sinus/superior orbital fissure.We are presenting a case report on THS. The patient presented with complete ptosis, unilateral headache and diminished vision. On examination there was complete ophthalmoplegia and diminished sensation over the area supplied by 1st and 2nd division of trigeminal nerve. CEMRI brain revealed enhancing soft tissue thickening along right cavernous sinus extending right orbital apex without any e/o filling defect in cavernous sinus or dilated SOV suggestive of THS. Other differential diagnosis were excluded on the basis of careful history, examination and investigations.

Keywords: cavernous sinus, Tolosa-Hunt syndrome, painful ophthalmoplegia, ptosis

[1]. Tolosa E. Periarteritic lesions of the carotid siphon with the clinical features of a carotid infraclinoid aneurysm. J Neurol Neurosurg Psychiatry 1954; 17:300–2.
[2]. Hunt WE, Meagher JN, Le Fever HE, et al. Painful ophthalmoplegia. Its relation to indolent inflammation of the cavernous sinus. Neurology 1961; 11:56–62.
[3]. Smith JL, Taxdal DSR. Painful ophthalmoplegia. The Tolosa-Hunt syndrome. Am J Ophthalmol 1966; 61:1466–72.
[4]. G. Iaconetta, L. Stella, M. Esposito, and P. Cappabianca, "Tolosa-Hunt syndrome extending in the cerebello-pontine angle," Cephalalgia, vol. 25, no. 9, pp. 746–750, 2005.
[5]. Pirastehfar M, Luo JJ. Tolosa-Hunt syndrome with facial nerve paresis. Neurological cases 2014; 2:13–16.

Abstract: Labor is defined as the process of regular uterine contraction leading to progressive effacement and dilatation of the cervix and birth of the baby. Induction of labor is an intervention designed to artificially initiate labor. Induction should only be considered when it is felt that the benefits of the delivery outweigh the potential maternal and fetal risks of awaiting spontaneous labor. Several methods are available for cervical ripening like prostaglandins, misoprostol, oxytocin, nitric oxide donors, mechanical methods like hygroscopic cervical dilators and amniotomy. The ideal agent is yet to be found .This is a prospective randomized controlled study conducted in the department of OBS and GYNAE in BABA SAHEB AMBEDKAR HOSPITAL............

[1]. Steiner AL, Creasy RK. Methods of cervical priming. Clin obstet Gynecol 1983; 26:37-46.
[2]. Calder AA. Nitric oxide-another factor in cervical ripening. Hum Reprod 1998; 13:250-1.
[3]. Chwalisz K, Shao-qing S, Garfield R, Beier H. Cervical ripening after a local application of nitric oxide. Hum Reprod 1997;12:2093-101.
[4]. Czerwenka K, et al. Human cervical ripening is associated with an increase in cervical inducible nitric oxide synthase expression. Biol Reprod 1999;60:1367-72.
[5]. Thomson AJ, Lunan CB, Ledingham M, Howart RC, Cameron IT, Greer IA, et al. Randomised trial of nitric oxide donor versus prostaglandin for cervical ripening before first-trimester termination of pregnancy. Lancet 1998; 352:1093-6.

Abstract: Introduction: Intrathecal anesthesia and epidural anesthesia (EA) are the most popular regional anesthesia techniques used for surgeries below umbilicus. EA is more versatile in providing anesthesia, analgesia and treatment of chronic disease syndromes. It provides better postoperative pain control and more rapid recovery from surgery. It also provides effective prolonged surgical anesthesia, prolonged postoperative analgesia, reduces the incidence of hemodynamic changes and reduces the incidence of PDPH as the dura is not pierced. Aims And Objectives: To Compare with the efficacy of lumbar epidural block with 0.5% bupivacaine 10ml with Fentanyl 50μg and 0.5%bupivacaine 10ml with butorphanol 1mg in lower limb surgeries focusing on Onset and duration of analgesia, Cardio respiratory effects, Sedation, Adverse effects......

Keywords: Epidural Anaesthesia, 0.5%Bupivacaine,Butorphanol,Fentanyl

[1]. RG Wheatley, SA Schug, D. Watson. Combination of local anaesthetic and an opioid combination. Br J Anaesth 2001;87:47
[2]. Modig J, Paalzow L. A comparison of epidural morphine and epidural bupivacaine for postoperative pain relief. Acta Anaesthesiol Scand 1981; 25: 437-41.
[3]. Abboud TK, Moore M, Zhu J, Murakawa K, Minehart M, Longhitano M et al. Epidural butorphanol for the relief of post-operative pain after caesarean section. Anaesthesiol 1986; 65: A397.
[4]. Tan PH, Chou AK, Perng JS, Chung HC, Lee CC, Mok MS. Comparison of epidural butorphanol plus clonidine with butorphanol alone for postoperative pain relief. Acta Anaesthesiol Sin 1997; 35: 91-6.
[5]. Gupta R, Kaur S, Singh S, Aujla KS. A comparison of Epidural Butorphanol and Tramadol for Postoperative Analgesia Using CSEA Technique. J Anaesthesiol Clin Pharmacol 2011; 27: 35-8.

Abstract: The term diabetes mellitus describes a complex metabolic disorder characterized by chronic hyper- glycemia resulting from defects in insulin secretion, insulin action or both. Inadequate insulin secretion and or diminished tissue responses to insulin in the complex pathways of hormone action results in deficient insulin action on target tissues, which leads to abnormalities of carbohydrate, fat, and protein metabolism. Impaired insulin secretion and or action may coexist in the same patient (1, 2). Most cases of diabetes can be classified into two broad etiopathogenetic categories : type 1 diabetes, which is characterized by an absolute deficiency of insulin secretion; or type 2 diabetes, which results from a combination of resistance to insulin action and an inadequate compensatory insulin secretory response.

[1]. Behrman R E, Kliegman R M, Jenson H B: Nelson Textbook of Pediatrics: Diabetes
[2]. Mellitus in Children. 171. thed. United States of America: Fletcher J; 2004. 1947.Fluckch E, Mullis PE. e diabetic child in the emergency room. er Umsch. 2005; 62(8):571-6.
[3]. Center for Disease Control (CDC). Hospitalizations for diabetic Ketoacidosis: Washington State, 1987-1989. MMR-Morb Mortal
[4]. Wkly Rep.1992; 41 (44): 837-9.Patel NR. Diabetic Ketoacidosis in the Pediatric Patient. Indian J Pediatr. 2002; 69 (1): 75-7.
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[6]. Saudi diabetics. Saudi Med J. 1987; 8(4):364 – 368.Balasubramanyam A, Zern JW, Hyman DJ, Pavlik V. New profiles of diabetic ketoacidosis: type I VS type 2 diabetes and the effect of ethnicity. Arch Intern Med. 1999; 159(19): 2317-22.Umpierrez GE, Kitabchi AE.

Abstract: Background: The aim of the present study was to determine and compare the shear bond strength of fifth, sixth, seventh and eighth generation dentin bonding agents on primary teeth. Materials and Method: Sixty extracted primary teeth were selected and assigned into four groups 1– fifth generation bonding agent (Tetric N Bond) (Total etch) (IVOCLAR), group 2– sixth generation (FL Bond II) (SHOFU), group 3– seventh generation (Tetric N Bond) (Self etch) (IVOCLAR) and group 4– eighth generation bonding agent (Tetric N Bond) (Universal) (IVOCLAR). With high speed handpiece coronal dentin was exposed. Selected dentin bonding agents were applied followed by composite restoration. All samples were saved in saline for 24 h and shear bond strength testing was done using universal testing machine.......

Keywords: Shear bond strength, primary teeth, self etch adhesive, total etch adhesive.

[1]. Buerkle V, Kuehnisch J, Guelmann M, Hickel R. Restoration materials for primary molars—results from a European survey. J Dent 2005;33:275–278.

[2]. Franzon R, Guimaraes LF, Magalhaes CE, Haas AN, Araujo FB. Outcomes of one-step incomplete and complete excavation in primary teeth: a 24-month randomized controlled trial. Caries Res 2014;48:376–383.

[3]. Swanson TK, Feigal RJ, Tantbirojn D, Hodges JS. Effect of adhesive systems and bevel on enamel margin integrity in primary and permanent teeth. Pediatr Dent 2008;30:134–140.

[4]. Donly KJ, Garcia Godoy F. The use of resin-based composite in children. Pediatr Dent 2002;24:480-488.

[5]. Mithiborwala S, Chaugule V, Munshi AK, Patil V. A comparison of the resin tag penetration of the total etch and the self-etch dentin bonding systems in the primary teeth: An in vitro study. Contemp Clin Dent 2012;3:158–163

Abstract: Pregnancy induced hypertension is characterized by abnormal blood pressure, proteinuria and edema that usually develops after the 20th week of pregnancy. The pregnant woman's body provides daily doses of 50 to 330 mg calcium to support the developing fetal skeleton. Early studies of blood calcium levels during pregnancy in humans found a significant decrease in the total serum calcium as pregnancy progressed. Regulation of intracellular calcium plays a key role in hypertension. Hypertension has been estimated to complicate 5% of all pregnancies and 11% of first pregnancies. Half of the pregnant women with hypertension have pre-eclampsia.

[1]. ACOG practice bulletin. Diagnosis and management of preeclampsia and eclampsia. Number 33, January 2002. Obstet Gynecol 2002; 99: 159-167
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[3]. Seely EW, Wood RJ and Brown EM et al., (1992). Lower serum ionized calcium and abnormal calcitrophic hormone levels in preeclampsia. Journal of Clinical Endocrinology and Metabolism 74(6) 1436-40.
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Abstract: The goal of periodontal therapy is to reconstruct the lost tissues to a functional state that is similar or identical to the tissue that has been lost due to disease process. One of the oldest method used as a scaffold is the fetal membranes. The fetal membrane posses various properties which gives light towards its use in periodontal regenerative therapy.This paper reviews the historical background, properties and the application of placental membranes in periodontics.

Keywords: Chorion, Amnion, Periodontics, Gingival Recession

[1]. Chakraborthy S, Sambashivaiah S, Kulal R, Bilchodmath S. Amnion and chorion allografts in combination with coronally advanced flap in the treatment of gingival recession: a clinical study. Journal of clinical and diagnostic research: JCDR. 2015 Sep;9(9):ZC98. [2]. Lafzi A, Farahani RM, Shoja MM, Tubbs RS. Amniotic membrane: A potential candidate for periodontal guided tissue regeneration?. Medical hypotheses. 2007 Jan 1;69(2):454.
[3]. Mermet I, Pottier N, Sainthillier JM, Malugani C, Cairey‐Remonnay S, Maddens S, Riethmuller D, Tiberghien P, Humbert P, Aubin F. Use of amniotic membrane transplantation in the treatment of venous leg ulcers. Wound repair and regeneration. 2007 Jul 1;15(4):459-64.
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[5]. Brindle A. Creation of an artificial vagina using ovular membranes of a term egg. Gynecol Obstet. 1934; 29: 385.

Abstract: The high prevalence of cardiovascular mortality in the end-stage renal disease population is well established. The aim of this current study was to document the relative prognostic significance of established biomarkers high-sensitive cardiac troponin T (hs cTnT), cardiac troponin I (cTnI), and N-terminal proBNP (NT-proBNP) and high-sensitive CRP (hs CRP) in this population. A prospective cohort study of dialysis patients undertaken in a single centre in Bulgaria. The relation between mortality and cardiac biomarkers was evaluated in 140 hemodialysis patients. End-point of interest was cardiac mortality. Statistical analysis using Cox proportional hazards was used to study relationship between competing covariates and outcome. Patients was followed upor a median duration of 24 months.......

Keywords: Cardiac biomarkers, hemodialysis, cardiac mortality

[1]. Hickman PE, McGill DA, Talaulikar G, Hiremagalur B, et al. Prognostic efficacy of cardiac biomarkers for mortality in dialysis patients. Intern Med J, 39(12), 2009, 812-8.
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[3]. Wang AY, Lai KN. Use of cardiac biomarkers in end-stage renal disease. J Am SocNephrol, 19(9), 2008, 1643-52.
[4]. Jacobs LH, van de Kerkhof J, Mingels AM, Kleijnen VW, et al. Haemodialysis patients longitudinally assessed by highly sensitive cardiac troponin T and commercial cardiac troponin T and cardiac troponin I assays. Ann ClinBiochem, 46(Pt 4), 2009, 283-90.
[5]. Heeschen C, Deu A, Langenbrink L, Goldmann BU, Hamm CW. Analytical and diagnostic performance of troponin assays in patients suspicious for acute coronary syndromes. Clin. Biochem, 33,2000, 359-368